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You Do Know Why They Call It Your Golden Years Don’t You?

Because you can spend the first half of your life ruining your health trying to accumulate riches, power and gold and then spend the last half of your life using all your gold to try and get your health back.

The good news is it doesn’t have to be that way at all.

If you’re not content with being another victim of industrial medicine . . . If you refuse to believe growing old is just another disease . . . If you believe life is the fruit of your inner most thoughts and not some game where you toss in your chips and leave, then AUDACIOUS AGING is for you.

Discover Why Dr. Andrew Weil Co-Authored Rose Cole’s Groundbreaking Book.


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Historical accounts for the search of the legendary “fountain of youth” can be traced back to Juan Ponce de León, Alexander the Great and even to Jerusalem when Jesus heals a man at the pool of Bethsaida.

I’ve heard more recently about the “Blue Zones” where people simply live longer and with less disease.

In conjunction with the National Institute on aging and top researchers in the field, Dan Buettner authored a book about four of these “Blue Zones:”

  1. Sardinia, Italy
  2. Okinawa, Japan
  3. Loma Linda, California
  4. Nicoya Peninsula, Costa Rica

Buettner found that people living in these communities shared common elements of lifestyle, diet and attitude. The most important things seem to be access to mineral rich water, mild exercise, low calorie diet and a personal sense of purpose.

From an evidence-based view point, reducing the consumption of dietary calories, or calorie restriction (CR) (1, 2) is the only intervention proven to extend both the average and maximum lifespan of all animal species tested.

Okinawans call the habit of calorie restriction, “Hara Hachi Bu,” which means to eat to 80% capacity.

Back in the lab, calorie restriction not only extends the lifespan of laboratory animals, but also reduces the incidence of virtually all diseases of aging such as cancer, (3,4,5,6) heart disease, (7,8) diabetes, (9,10,11) osteoporosis,(12,13) auto-immune disorders, (14,15,16) neurological decline (17,18) and diseases such as Alzheimer’s (19) and Parkinson’s (20,21,22).

Those references will take you to clinical abstracts at the National Library of Medicine, I encourage you to follow them to learn more about how you can slow aging, prevent and possibly cure degenerative dis-eases, naturally.

Obviously, the quality of food and how it’s prepared is of the utmost importance, as well. If your fruits and vegetables are organically harvested from fertile, mineral rich soils you will be better off, than eating herbicide tainted, hormone injected, radiated and genetically manufactured plants and animals.

Two-time Nobel Prize laureate Dr. Linus Pauling believed 90% of dis-ease is caused from mineral deficiencies. Life giving minerals come from pristine water sources, as well from healthy plants and herbaceous animals we consume.

So, eating right and eating less isn’t only smart, like mild exercise and fresh air and water, it should be common sense.

In 1985 a special report was published by the National Institute of Health, Centers for Disease Control and the Department of Health and Human Services stating that the best body weight associated with the greatest longevity are generally below the average weight of the U.S. population (23).

This information was gathered by insurance companies, the American Cancer Society and other long-term studies like the Framington Heart Study and the Manitoba study.

Eight years later the Journal of the American Medical Association further concluded that body weight and death rates were directly related.

Without exception every developing country that embraces the same “fast food culture”  and Standard American Diet  (SAD) that originated in America during the 1950’s, also experience the same increased rates of obesity, dis-ease and premature death by diet that the U.S. did over the last 70 years (24).

P.S. Science is finally explaining why so many people are slave to pharmaceuticals (50%) and why 40% of senior income goes into medical costs such as drugs. Frankly, only YOU can prevent accelerated aging and related dis-eases.


Refs:
[1] Taubes, G. (2000). The Famine of Youth. Scientific American, June.
[2] Study that discovered calorie restriction extends animal lifespan: McCay CM, et al. (1935). The effect of retarded growth upon the length of life span and upon the ultimate body size. Journal of Nutrition, 10(1), pages 63-79.
[3] Kritchevsky D, & Klurfeld DM. (1986). Influence of caloric intake on experimental carcinogenesis: a review. Advances in Experimental Medicine & Biology, 206, pages 55-68.
[4] Albanes D. (1987) Caloric intake, body weight, and cancer: a review. Nutrition & Cancer, 9(4), pages 199-217.
[5] Kritchevsky D. (1997). Caloric restriction and experimental mammary carcinogenesis. Breast Cancer Research & Treatment, Nov-Dec, 46(2-3), pages 161-7.
[6] Kritchevsky D. (2001). Caloric restriction and cancer. Journal of Nutritional Science and Vitaminology (Tokyo). February, 47(1), page 13-9.
[7] Swoap SJ. (2001). Altered leptin signaling is sufficient, but not required, for hypotension associated with caloric restriction. American Journal of Physiology, Heart & Circulatory Physiology, December, 281(6):H2473-9.
[8] Keenan KP, et al (1994). The effects of overfeeding and dietary restriction on Sprague-Dawley rat survival and early pathology biomarkers of aging. Toxicologic Pathology, May-June, 22(3), pages 300-15.
[9] Stern JS, et at. (2001). Calorie restriction in obesity: prevention of kidney disease in rodents. Journal of Nutrition, March, 131(3), pages 913S-917S.
[10] Fujioka K, et al (2000). Weight loss with sibutramine improves glycaemic control and other metabolic parameters in obese patients with type 2 diabetes mellitus. Diabetes, Obesity & Metabolism, June, 2(3), pages 175-87.
[11] Okauchi N, et al. (1995). Is caloric restriction effective in preventing diabetes mellitus in the Otsuka Long Evans Tokushima fatty rat, a model of spontaneous non-insulin-dependent diabetes mellitus? Diabetes Research and Clinical Practice, February, 27(2), pages 97-106.
[12] Kalu DN. (1984). Aging and dietary modulation of rat skeleton and parathyroid hormone. Endocrinology, October, 115(4), pages 1239-47.
[13] Kalu DN. (1984). Lifelong food restriction prevents senile osteopenia and hyperparathyroidism in F344 rats. Mechanisms of Ageing and Development, July, 26(1), pages 103-12.
[14] Fernandes G. (1983). Influence of diet on vascular lesions in autoimmune-prone B/W mice. Proceedings of the National Academy of Sciences, February, 80(3), pages 874-7.
[15] Nandy K. (1982). Effects of controlled dietary restriction on brain-reactive antibodies in sera of aging mice. Mechanisms of Ageing & Development, February, 18(2), pages 97-102.
[16] Fernandes G, et al. (1976). Influence of diet on survival of mice. Proceedings of the National Academy of Sciences, April, 73(4), pages 1279-83. Neurological decline
[17] Means LW, et al.(1993). Mid-life onset of dietary restriction extends life and prolongs cognitive functioning. Physiology & Behavior, September, 54(3), pages 503-8.
[18] Pitsikas N, & Algeri S. (1992). Deterioration of spatial and nonspatial reference and working memory in aged rats: protective effect of life-long calorie restriction. Neurobiology of Aging, May-Jun, 13(3), pages 369-73.
[19] Mattson MP. (2000). Existing data suggest that Alzheimer’s disease is preventable. Annals of the New York Academy of Sciences, 924, pages 153-9.
[20] Roth GS, et al. (1984). Delayed loss of striatal dopamine receptors during aging of dietarily restricted rats. Brain Research, May 21, 300(1), pages 27-32.
[21] Levin P, et al. (1981). Dietary restriction retards the age-associated loss of rat striatal dopaminergic receptors. Science, October 30, 214(4520), pages 561-2.
[22] Ingram DK, et al. (1987). Dietary restriction benefits learning and motor performance of aged mice. Journal of Gerontology, January, 42(1), pages 78-81.
[23] NIHNC, CDC, & DHHS. (1985). Body weight, health and longevity: conclusions and recommendations of the workshop. Nutrition Reviews, February, 43(2), pages 61-3.
[24] Lee IM. et al. (1993). Body weight and mortality. A 27-year follow-up of middle-aged men. Journal of the American Medical Association, December 15, 270(23), pages 2823-8.

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